This study investigates the protective effects of N-acetylcysteine (NAC) in mitigating ovarian failure induced by doxorubicin (DOX), a chemotherapeutic agent known for its detrimental effects on female reproductive health. While chemotherapy provides life-saving cancer treatment, it often leads to severe reproductive toxicity, including damage to ovarian follicles and premature ovarian failure. This research explores NAC’s potential as a therapeutic agent by focusing on its antioxidant properties, which may counteract oxidative stress and inflammation, two primary mechanisms of DOX-induced ovarian damage. Through a series of histopathological, and biochemical, the study provides evidence that NAC preserves ovarian function and structure, offering a promising approach to protecting fertility in women undergoing chemotherapy.

The Future of Scientific Publishing: Trends and Innovations
Introduction: Scientific publishing is constantly evolving, driven by technological advancements and changing research practices. This blog post explores the latest trends and innovations shaping the